A recent study has revealed the metabolic pathway that bacteria use to survive in bone, which could potentially act as a target for the development of new antimicrobial compounds.
New research has suggested that a protein involved in Alzheimer’s disease may also be implicated in HIV-associated neurological disorders, suggesting Alzheimer’s therapies may benefit a subset of HIV patients.
In our latest infographic we provide a visual snapshot of the infections and immune processes that have been linked with the microbiome thus far, explaining some of the evidence
This study, recently published in Future Microbiology, aimed to study the effects of encoded heat-shock protein 90 homolog (htpG) on the selected virulence factors responsible for pathogenesis and biofilm formation of P. aeruginosa.
Researchers have revealed that frequent recurrences of MRSA infection in mouse models may be due to bacterial toxins that ‘kill’ lymphatic muscle cells, effectively impairing their ability to pump lymphatic fluid to the lymph nodes.
This research, published in Future Microbiology, screens host proteins for those that interact with EspF via flow cytometry and high-throughput screening to try and shed light on how EspF affects host cells to induce colitis and even colorectal carcinogenesis.
The exact mechanism(s) linking fetal Zika infection and congenital microcephaly remains unclear, however, an international team of researchers have now identified a possible role for unfolded protein responses in this process.
Scientists have revealed proteins, termed RIFINs, expressed on the surface of infected erythrocytes that may help the malaria parasite to suppress the host immune response, leading to severe infection.
This study looks at Candida tropicalis, evaluating if radiation used in radiotherapy can cause changes in the virulence potential.
In a recently published study, researchers have utilized spectroscopy techniques to provide an in-depth understanding of the atomic interaction that occurs when viral buds are released from the host cell’s membrane after infection.
